You seem to have a more in depth grasp of the precise genetics involved here than I do, what would your opinion be of Dr. Frye’s concept of “Cerebral Folate Disorder” that I mention in another comment?
Here’s a paper from him and his team, he has many though:
As best I can tell, he is focused on the non-syndromic, non-verbal, uh, what this recent paper OP is about seems to categorize as ‘early diagnosis autism’.
He’s got a cluster of specific mutations that produce an evidenced, differing neurochemistry in the brain, and apparently a potential treatment for that ‘subypr/component’ as well?
I… don’t agree with his general description of autism as basically only the kind that makes you developmentally delayed, but, if you can get past that… do you think he may be onto something as far as that being an distinct ‘type’ of autism?
Also, apologies if I am using some terms incorrectly or innacurately, I am not a neuroscientist.
You seem to have a more in depth grasp of the precise genetics involved here than I do, what would your opinion be of Dr. Frye’s concept of “Cerebral Folate Disorder” that I mention in another comment?
Here’s a paper from him and his team, he has many though:
https://pmc.ncbi.nlm.nih.gov/articles/PMC5794882/
As best I can tell, he is focused on the non-syndromic, non-verbal, uh, what this recent paper OP is about seems to categorize as ‘early diagnosis autism’.
He’s got a cluster of specific mutations that produce an evidenced, differing neurochemistry in the brain, and apparently a potential treatment for that ‘subypr/component’ as well?
I… don’t agree with his general description of autism as basically only the kind that makes you developmentally delayed, but, if you can get past that… do you think he may be onto something as far as that being an distinct ‘type’ of autism?
Also, apologies if I am using some terms incorrectly or innacurately, I am not a neuroscientist.